Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 293 entries
Sorted by: Best Match Show Resources per page
Nanomedicine: Application of nanoparticles in clinical therapies and diagnostics.

The international journal of biochemistry & cell biology

Kargul J, Irminger-Finger I, Laurent GJ.
PMID: 27130962
Int J Biochem Cell Biol. 2016 Jun;75:140. doi: 10.1016/j.biocel.2016.04.011. Epub 2016 Apr 27.

No abstract available.

Cite
Kargul J, Irminger-Finger I, Laurent GJ. Nanomedicine: Application of nanoparticles in clinical therapies and diagnostics. Int J Biochem Cell Biol. 2016;75:140doi: 10.1016/j.biocel.2016.04.011.
Kargul, J., Irminger-Finger, I., & Laurent, G. J. (2016). Nanomedicine: Application of nanoparticles in clinical therapies and diagnostics. The international journal of biochemistry & cell biology, 75140. https://doi.org/10.1016/j.biocel.2016.04.011
Kargul, Joanna, et al. "Nanomedicine: Application of nanoparticles in clinical therapies and diagnostics." The international journal of biochemistry & cell biology vol. 75 (2016): 140. doi: https://doi.org/10.1016/j.biocel.2016.04.011
Kargul J, Irminger-Finger I, Laurent GJ. Nanomedicine: Application of nanoparticles in clinical therapies and diagnostics. Int J Biochem Cell Biol. 2016 Jun;75:140. doi: 10.1016/j.biocel.2016.04.011. Epub 2016 Apr 27. PMID: 27130962.
Copy Download .nbib Format: NLM
WITHDRAWN: The nonstructural protein-1 of influenza virus A.

The international journal of biochemistry & cell biology

Jiang H, Xu C, Zhang Y, Chen G, Xiao Y.
PMID: 19013254
Int J Biochem Cell Biol. 2008 Nov 01; doi: 10.1016/j.biocel.2008.10.016. Epub 2008 Nov 01.

This article has been retracted; please see Elsevier Policy on Article Withdrawal: http://www.elsevier.com/locate/withdrawalpolicy This article has been retracted at the request of the Editor-in-Chief of The International Journal of Biochemistry and Cell Biology, as the authors have plagiarised sections...

Cite
Jiang H, Xu C, Zhang Y, et al. WITHDRAWN: The nonstructural protein-1 of influenza virus A. Int J Biochem Cell Biol. 2008;doi: 10.1016/j.biocel.2008.10.016.
Jiang, H., Xu, C., Zhang, Y., Chen, G., & Xiao, Y. (2008). WITHDRAWN: The nonstructural protein-1 of influenza virus A. The international journal of biochemistry & cell biology, . https://doi.org/10.1016/j.biocel.2008.10.016
Jiang, Hua, et al. "WITHDRAWN: The nonstructural protein-1 of influenza virus A." The international journal of biochemistry & cell biology vol. (2008). doi: https://doi.org/10.1016/j.biocel.2008.10.016
Jiang H, Xu C, Zhang Y, Chen G, Xiao Y. WITHDRAWN: The nonstructural protein-1 of influenza virus A. Int J Biochem Cell Biol. 2008 Nov 01; doi: 10.1016/j.biocel.2008.10.016. Epub 2008 Nov 01. PMID: 19013254.
Copy Download .nbib Format: NLM
Frontiers in diabetic research.

The international journal of biochemistry & cell biology

Leung PS, Hayek A, de Gasparo M.
PMID: 16481209
Int J Biochem Cell Biol. 2006;38(5):687-8. doi: 10.1016/j.biocel.2006.01.012. Epub 2006 Feb 02.

No abstract available.

Cite
Leung PS, Hayek A, de Gasparo M. Frontiers in diabetic research. Int J Biochem Cell Biol. 2006;38(5):687-8doi: 10.1016/j.biocel.2006.01.012.
Leung, P. S., Hayek, A., & de Gasparo, M. (2006). Frontiers in diabetic research. The international journal of biochemistry & cell biology, 38(5), 687-8. https://doi.org/10.1016/j.biocel.2006.01.012
Leung, Po Sing, et al. "Frontiers in diabetic research." The international journal of biochemistry & cell biology vol. 38,5 (2006): 687-8. doi: https://doi.org/10.1016/j.biocel.2006.01.012
Leung PS, Hayek A, de Gasparo M. Frontiers in diabetic research. Int J Biochem Cell Biol. 2006;38(5):687-8. doi: 10.1016/j.biocel.2006.01.012. Epub 2006 Feb 02. PMID: 16481209.
Copy Download .nbib Format: NLM
Corrigendum to 'specific changes in the proteomic pattern produced by the BRCA1-Ser1841Asn missense mutation' [International Journal of Biochemistry and Cell Biology (2007) 220-226].

The international journal of biochemistry & cell biology

Crugliano T, Quaresima B, Gaspari M, Faniello MC, Romeo F, Baudi F, Cuda G, Costanzo F, Venuta S.
PMID: 28351545
Int J Biochem Cell Biol. 2017 Jul;88:236-237. doi: 10.1016/j.biocel.2017.03.006. Epub 2017 Mar 25.

No abstract available.

Cite
Crugliano T, Quaresima B, Gaspari M, et al. Corrigendum to 'specific changes in the proteomic pattern produced by the BRCA1-Ser1841Asn missense mutation' [International Journal of Biochemistry and Cell Biology (2007) 220-226]. Int J Biochem Cell Biol. 2017;88:236-237doi: 10.1016/j.biocel.2017.03.006.
Crugliano, T., Quaresima, B., Gaspari, M., Faniello, M. C., Romeo, F., Baudi, F., Cuda, G., Costanzo, F., & Venuta, S. (2017). Corrigendum to 'specific changes in the proteomic pattern produced by the BRCA1-Ser1841Asn missense mutation' [International Journal of Biochemistry and Cell Biology (2007) 220-226]. The international journal of biochemistry & cell biology, 88236-237. https://doi.org/10.1016/j.biocel.2017.03.006
Crugliano, Telma, et al. "Corrigendum to 'specific changes in the proteomic pattern produced by the BRCA1-Ser1841Asn missense mutation' [International Journal of Biochemistry and Cell Biology (2007) 220-226]." The international journal of biochemistry & cell biology vol. 88 (2017): 236-237. doi: https://doi.org/10.1016/j.biocel.2017.03.006
Crugliano T, Quaresima B, Gaspari M, Faniello MC, Romeo F, Baudi F, Cuda G, Costanzo F, Venuta S. Corrigendum to 'specific changes in the proteomic pattern produced by the BRCA1-Ser1841Asn missense mutation' [International Journal of Biochemistry and Cell Biology (2007) 220-226]. Int J Biochem Cell Biol. 2017 Jul;88:236-237. doi: 10.1016/j.biocel.2017.03.006. Epub 2017 Mar 25. PMID: 28351545.
Copy Download .nbib Format: NLM
Biochemical characterization and mutational analysis of a novel flap endonuclease 1 from Thermococcus barophilus Ch5.

The international journal of biochemistry & cell biology

Lin T, Zhang L, Jiang D, Wu L, Chen K, Li L, Jin C, Li Z, Oger P.
PMID: 34990837
Int J Biochem Cell Biol. 2022 Jan 03;143:106154. doi: 10.1016/j.biocel.2021.106154. Epub 2022 Jan 03.

Flap endonuclease 1 (FEN1) plays important roles in DNA replication, repair and recombination. Herein, we report biochemical characteristics and catalytic mechanism of a novel FEN1 from the hyperthermophilic euryarchaeon Thermococcus barophilus Ch5 (Tb-FEN1). As expected, the recombinant Tb-FEN1 can...

Cite
Lin T, Zhang L, Jiang D, et al. Biochemical characterization and mutational analysis of a novel flap endonuclease 1 from Thermococcus barophilus Ch5. Int J Biochem Cell Biol. 2022;143:106154doi: 10.1016/j.biocel.2021.106154.
Lin, T., Zhang, L., Jiang, D., Wu, L., Chen, K., Li, L., Jin, C., Li, Z., & Oger, P. (2022). Biochemical characterization and mutational analysis of a novel flap endonuclease 1 from Thermococcus barophilus Ch5. The international journal of biochemistry & cell biology, 143106154. https://doi.org/10.1016/j.biocel.2021.106154
Lin, Tan, et al. "Biochemical characterization and mutational analysis of a novel flap endonuclease 1 from Thermococcus barophilus Ch5." The international journal of biochemistry & cell biology vol. 143 (2022): 106154. doi: https://doi.org/10.1016/j.biocel.2021.106154
Lin T, Zhang L, Jiang D, Wu L, Chen K, Li L, Jin C, Li Z, Oger P. Biochemical characterization and mutational analysis of a novel flap endonuclease 1 from Thermococcus barophilus Ch5. Int J Biochem Cell Biol. 2022 Jan 03;143:106154. doi: 10.1016/j.biocel.2021.106154. Epub 2022 Jan 03. PMID: 34990837.
Copy Download .nbib Format: NLM
[No title available]

Biochemistry and cell biology = Biochimie et biologie cellulaire

Wang Y, Sun L, Wang L, Yu H, Yu X, Zou Y.
PMID: 34734756
Biochem Cell Biol. 2021 Dec;99(6):735-740. doi: 10.1139/bcb-2021-0044. Epub 2021 Nov 04.

Preeclampsia is a severe pregnancy complication characterized by hypertension and may cause maternal morbidity and mortality. A better understanding of the essential genes involved in preeclampsia pathophysiology is urgently needed. This study investigated the function and molecular mechanisms of...

Cite
Wang Y, Sun L, Wang L, et al. . Biochem Cell Biol. 2021;99(6):735-740doi: 10.1139/bcb-2021-0044.
Wang, Y., Sun, L., Wang, L., Yu, H., Yu, X., & Zou, Y. (2021). . Biochemistry and cell biology = Biochimie et biologie cellulaire, 99(6), 735-740. https://doi.org/10.1139/bcb-2021-0044
Wang, Yuping, et al. "." Biochemistry and cell biology = Biochimie et biologie cellulaire vol. 99,6 (2021): 735-740. doi: https://doi.org/10.1139/bcb-2021-0044
Wang Y, Sun L, Wang L, Yu H, Yu X, Zou Y. . Biochem Cell Biol. 2021 Dec;99(6):735-740. doi: 10.1139/bcb-2021-0044. Epub 2021 Nov 04. PMID: 34734756.
Copy Download .nbib Format: NLM
The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy.

The international journal of biochemistry & cell biology

Chidipi B, Angulo MB, Shah SI, Rieser M, Ullah G, McDonald TV, Noujaim SF.
PMID: 34923139
Int J Biochem Cell Biol. 2021 Dec 16;143:106137. doi: 10.1016/j.biocel.2021.106137. Epub 2021 Dec 16.

Friedreich ataxia is an autosomal recessive congenital neurodegenerative disease caused by a deficiency in the frataxin protein and is often diagnosed in young adulthood. An expansion of guanine-adenine-adenine repeats in the first intron of the FXN gene leads to...

Cite
Chidipi B, Angulo MB, Shah SI, et al. The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy. Int J Biochem Cell Biol. 2021;143:106137doi: 10.1016/j.biocel.2021.106137.
Chidipi, B., Angulo, M. B., Shah, S. I., Rieser, M., Ullah, G., McDonald, T. V., & Noujaim, S. F. (2021). The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy. The international journal of biochemistry & cell biology, 143106137. https://doi.org/10.1016/j.biocel.2021.106137
Chidipi, Bojjibabu, et al. "The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy." The international journal of biochemistry & cell biology vol. 143 (2021): 106137. doi: https://doi.org/10.1016/j.biocel.2021.106137
Chidipi B, Angulo MB, Shah SI, Rieser M, Ullah G, McDonald TV, Noujaim SF. The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy. Int J Biochem Cell Biol. 2021 Dec 16;143:106137. doi: 10.1016/j.biocel.2021.106137. Epub 2021 Dec 16. PMID: 34923139.
Copy Download .nbib Format: NLM
Deletion of ribosomal paralogs Rpl39 and Rpl39l compromises cell proliferation via protein synthesis and mitochondrial activity.

The international journal of biochemistry & cell biology

Zou Q, Qi H.
PMID: 34428590
Int J Biochem Cell Biol. 2021 Oct;139:106070. doi: 10.1016/j.biocel.2021.106070. Epub 2021 Aug 21.

Accumulating evidences suggest that the composition and functional roles of ribosomes are heterogeneous in cells, partly due to the temporal-spatial expression of paralogous ribosomal proteins (RPs), of which functional relationships remain largely unexplored. In mouse, the X chromosome-linked RPL39...

Cite
Zou Q, Qi H. Deletion of ribosomal paralogs Rpl39 and Rpl39l compromises cell proliferation via protein synthesis and mitochondrial activity. Int J Biochem Cell Biol. 2021;139:106070doi: 10.1016/j.biocel.2021.106070.
Zou, Q., & Qi, H. (2021). Deletion of ribosomal paralogs Rpl39 and Rpl39l compromises cell proliferation via protein synthesis and mitochondrial activity. The international journal of biochemistry & cell biology, 139106070. https://doi.org/10.1016/j.biocel.2021.106070
Zou, Qianxing, and Qi, Huayu. "Deletion of ribosomal paralogs Rpl39 and Rpl39l compromises cell proliferation via protein synthesis and mitochondrial activity." The international journal of biochemistry & cell biology vol. 139 (2021): 106070. doi: https://doi.org/10.1016/j.biocel.2021.106070
Zou Q, Qi H. Deletion of ribosomal paralogs Rpl39 and Rpl39l compromises cell proliferation via protein synthesis and mitochondrial activity. Int J Biochem Cell Biol. 2021 Oct;139:106070. doi: 10.1016/j.biocel.2021.106070. Epub 2021 Aug 21. PMID: 34428590.
Copy Download .nbib Format: NLM
TRIM58 inactivates p53/p21 to promote chemoresistance via ubiquitination of DDX3 in breast cancer.

The international journal of biochemistry & cell biology

Wang J, Yang F, Zhuang J, Huo Q, Li J, Xie N.
PMID: 34954155
Int J Biochem Cell Biol. 2021 Dec 23;143:106140. doi: 10.1016/j.biocel.2021.106140. Epub 2021 Dec 23.

Chemotherapy resistance is that the most important reason behind of carcinoma treatment failure but the underlying molecular mechanisms are unclear. Members of the tripartite motifcontaining protein (TRIM) family play crucial roles in the carcinogenesis and development of resistance against...

Cite
Wang J, Yang F, Zhuang J, et al. TRIM58 inactivates p53/p21 to promote chemoresistance via ubiquitination of DDX3 in breast cancer. Int J Biochem Cell Biol. 2021;143:106140doi: 10.1016/j.biocel.2021.106140.
Wang, J., Yang, F., Zhuang, J., Huo, Q., Li, J., & Xie, N. (2021). TRIM58 inactivates p53/p21 to promote chemoresistance via ubiquitination of DDX3 in breast cancer. The international journal of biochemistry & cell biology, 143106140. https://doi.org/10.1016/j.biocel.2021.106140
Wang, Juan, et al. "TRIM58 inactivates p53/p21 to promote chemoresistance via ubiquitination of DDX3 in breast cancer." The international journal of biochemistry & cell biology vol. 143 (2021): 106140. doi: https://doi.org/10.1016/j.biocel.2021.106140
Wang J, Yang F, Zhuang J, Huo Q, Li J, Xie N. TRIM58 inactivates p53/p21 to promote chemoresistance via ubiquitination of DDX3 in breast cancer. Int J Biochem Cell Biol. 2021 Dec 23;143:106140. doi: 10.1016/j.biocel.2021.106140. Epub 2021 Dec 23. PMID: 34954155.
Copy Download .nbib Format: NLM
GCN5 enables HSP12 induction promoting chromatin remodeling, not histone acetylation.

Biochemistry and cell biology = Biochimie et biologie cellulaire

Antonazzi F, Di Felice F, Camilloni G.
PMID: 34102063
Biochem Cell Biol. 2021 Dec;99(6):700-706. doi: 10.1139/bcb-2020-0620. Epub 2021 Jun 08.

Regulation of stress-responsive genes represent one of the best examples of gene induction, and the relevance and involvement of different regulators may change for a given gene depending on the challenging stimulus. The

Cite
Antonazzi F, Di Felice F, Camilloni G. GCN5 enables HSP12 induction promoting chromatin remodeling, not histone acetylation. Biochem Cell Biol. 2021;99(6):700-706doi: 10.1139/bcb-2020-0620.
Antonazzi, F., Di Felice, F., & Camilloni, G. (2021). GCN5 enables HSP12 induction promoting chromatin remodeling, not histone acetylation. Biochemistry and cell biology = Biochimie et biologie cellulaire, 99(6), 700-706. https://doi.org/10.1139/bcb-2020-0620
Antonazzi, Francesca, et al. "GCN5 enables HSP12 induction promoting chromatin remodeling, not histone acetylation." Biochemistry and cell biology = Biochimie et biologie cellulaire vol. 99,6 (2021): 700-706. doi: https://doi.org/10.1139/bcb-2020-0620
Antonazzi F, Di Felice F, Camilloni G. GCN5 enables HSP12 induction promoting chromatin remodeling, not histone acetylation. Biochem Cell Biol. 2021 Dec;99(6):700-706. doi: 10.1139/bcb-2020-0620. Epub 2021 Jun 08. PMID: 34102063.
Copy Download .nbib Format: NLM
Isovitexin attenuates tumor growth in human colon cancer cells through the modulation of apoptosis and epithelial-mesenchymal transition via PI3K/Akt/mTOR signaling pathway.

Biochemistry and cell biology = Biochimie et biologie cellulaire

Zhu H, Zhao N, Jiang M.
PMID: 34219464
Biochem Cell Biol. 2021 Dec;99(6):741-749. doi: 10.1139/bcb-2021-0045. Epub 2021 Jul 04.

Isovitexin, a biologically active flavone C-glycosylated derivative, has a variety of biological activities. We aimed to identify the effect of isovitexin (Isov) on colon cancer. Human colonic epithelial cells (HCECs) and cancer cells were treated with Isov and Cell...

Cite
Zhu H, Zhao N, Jiang M. Isovitexin attenuates tumor growth in human colon cancer cells through the modulation of apoptosis and epithelial-mesenchymal transition via PI3K/Akt/mTOR signaling pathway. Biochem Cell Biol. 2021;99(6):741-749doi: 10.1139/bcb-2021-0045.
Zhu, H., Zhao, N., & Jiang, M. (2021). Isovitexin attenuates tumor growth in human colon cancer cells through the modulation of apoptosis and epithelial-mesenchymal transition via PI3K/Akt/mTOR signaling pathway. Biochemistry and cell biology = Biochimie et biologie cellulaire, 99(6), 741-749. https://doi.org/10.1139/bcb-2021-0045
Zhu, Hao, et al. "Isovitexin attenuates tumor growth in human colon cancer cells through the modulation of apoptosis and epithelial-mesenchymal transition via PI3K/Akt/mTOR signaling pathway." Biochemistry and cell biology = Biochimie et biologie cellulaire vol. 99,6 (2021): 741-749. doi: https://doi.org/10.1139/bcb-2021-0045
Zhu H, Zhao N, Jiang M. Isovitexin attenuates tumor growth in human colon cancer cells through the modulation of apoptosis and epithelial-mesenchymal transition via PI3K/Akt/mTOR signaling pathway. Biochem Cell Biol. 2021 Dec;99(6):741-749. doi: 10.1139/bcb-2021-0045. Epub 2021 Jul 04. PMID: 34219464.
Copy Download .nbib Format: NLM
The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy.

The international journal of biochemistry & cell biology

Chidipi B, Angulo MB, Shah SI, Rieser M, Ullah G, McDonald TV, Noujaim SF.
PMID: 34923139
Int J Biochem Cell Biol. 2021 Dec 16;143:106137. doi: 10.1016/j.biocel.2021.106137. Epub 2021 Dec 16.

Friedreich ataxia is an autosomal recessive congenital neurodegenerative disease caused by a deficiency in the frataxin protein and is often diagnosed in young adulthood. An expansion of guanine-adenine-adenine repeats in the first intron of the FXN gene leads to...

Cite
Chidipi B, Angulo MB, Shah SI, et al. The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy. Int J Biochem Cell Biol. 2021;143:106137doi: 10.1016/j.biocel.2021.106137.
Chidipi, B., Angulo, M. B., Shah, S. I., Rieser, M., Ullah, G., McDonald, T. V., & Noujaim, S. F. (2021). The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy. The international journal of biochemistry & cell biology, 143106137. https://doi.org/10.1016/j.biocel.2021.106137
Chidipi, Bojjibabu, et al. "The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy." The international journal of biochemistry & cell biology vol. 143 (2021): 106137. doi: https://doi.org/10.1016/j.biocel.2021.106137
Chidipi B, Angulo MB, Shah SI, Rieser M, Ullah G, McDonald TV, Noujaim SF. The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich's ataxia cardiomyopathy. Int J Biochem Cell Biol. 2021 Dec 16;143:106137. doi: 10.1016/j.biocel.2021.106137. Epub 2021 Dec 16. PMID: 34923139.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 293 entries